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Albert proposed a humorous conclusion to his study, but one which surely has some truth to it. The battle between the utilitarian and the emotional is not exclusive to abstract dilemmas. Actually, these two systems are invariably expressed in almost all of our deliberations. We are in general more aggressive, sometimes violently and mercilessly, with those we love most. Within the trust of an unvarnished, unprejudiced relationship with vast expectations, jealousy, fatigue and pain, sometimes irrational rage emerges. The same argument between a couple that seems unbearable when we are living through it becomes insignificant and often ridiculous when seen from a third-person perspective. The answer is that the consideration is not utilitarian but rather capriciously deontological. The deontology threshold drops precipitously and we are not willing to make the slightest effort to resolve something that would alleviate all the tension. And Albert, half joking and half serious, suggests that the next time we are fighting with our significant other, we should switch to Spanish (or any other non-native language). This would allow us to bring the argument into a more rational arena, one less burdened by visceral epithets. In many cases, acting pragmatically or in a utilitarian manner requires detaching ourselves from strong emotional convictions. And it implies (most often implicitly) assigning a value (or a prize) to issues that, from a deontological perspective, it seems impossible to rationalize and convert into numbers. You are driving and right after you have crossed a railway line you realize that the warning signs at the level crossing are not working. But you understand that with the traffic due to get heavier someone will be hit by a train and most likely die. Would you hang up and let someone die to gain a few minutes and make it on time to your meeting It does not matter if it is one, five, ten or one million people arriving late, the answer is the same.
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Tese ticipate in the learning of skills and habits, especially those systems support diferent aspects of memory, and their that require implicit motor learning. Sensory in human amnesic patients, in animals, and in normal volun registration, perceptual representation, short-term and ters using electrophysiological and neuroimaging methods working memory, procedural memory, semantic memory, permit us to elaborate on the cognitive model frst presented and episodic memory all represent systems or subsystems in Figure 9. The brain structures that sup neural systems that support the memory functions listed port various memory processes difer, depending on the (Figure 9. At the cellular level, changes in the synaptic together the relationships among diferent tpes of infor strengths bet n neurons in neural netorks in the medial mation about an episode; (b) the parietal cortex, which is temporal lobe, neocortex, cerebellum, and elsewhere are the involved in the encoding and retrieving of episodic or con most likely mechanisms for learning and memory. Bit by bit, textual memory; (c) the prefontal cortex, which is involved we are developing a very clear understanding of the molecu in encoding and retrieving information based perhaps on lar processes that support synaptic plasticit, and thus learn the nature of the material being processed; (d) the temporal ing and memory, in the brain. Do you think that, if you gave a detailed set of on the basis of their time course. Does the fact that instructions to another person who had never ridden some memories last seconds while some last a lifetime a bike, she could carefully study your instructions and necessarily imply that diferent neural systems mediate then hop on a bike and happily ride of into the sunset What can they learn, and what does this tell us about how constraints do cognitive neuroscience fndings place on memories are encoded in the brain Can you describe to others the principles of riding Suggested Reading Aggleton, J. Mark Twain When dealing with people, remember you are not dealing with creatures of logic, but creatures of emotion. This was not because she had not ben in fightening circumstances; in fact, she had ben in plent. She had ben held at both knife and gunpoint, physically accosted by a woman tice her size, and nearly killed in a domestic violence atack, among other experiences (Feinstein et al. The deterioration of her amygdalae was Theories of Emotion Generation highly specifc; surrounding white mater showed minimal damage. On stan dard neuropsychological tests, her intelligence scores were in the normal range, the Amygdala and she had no perceptual or motor problems. Something curious popped up, Interactions Between Emotion and however, when her emotional processing was tested. But one facial expression Emotion stumped her: fear (se the botom right photo of Figure 10. When asked to draw pictures depicting diferent emotions, she was able to provide reasonable cartoons of a range of states, except when asked to depict fear.
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A sportsman presents having sprained C Continue sport, by controlling symptoms his thumb. B Grade 1 (c) Give two reasons why non-steroidal C Grade 2 anti-inflammatories might be helpful. D You cannot tell until you have compared A To reduce pain it with the other side. B To reduce oedema C To enable immediate return to sport Bursae before the injury is healed 5. D To prevent the problem spreading to the A They are normal structures designed to other leg. B They are lined with synovial membrane Soft-tissue haematoma and connected to the joint beneath. Which of the following statements C They are able to become inflamed and regarding soft-tissue haematomas are infected. D the bruised muscle may sometimes be A the first phase of bone healing is the replaced with cartilage. A 50 per cent D Stress on bone stimulates more bone to B 50 per cent be laid down. A Are most common in high intensity high A 50 per cent load sports B 50 per cent B Produce highly localised pain C 80 per cent. What would you offer him as the chances of the repair being Ankle injury successful A rugby player fell onto his shoulder B To allow full strength in the ankle some weeks ago. He now presents C To improve proprioception with a painful lump about 4 cm in D To avoid premature onset of diameter on the top of his shoulder. B the acromioclavicular joint C the head of the humerus Injuries to the plantarflexor D the sternoclavicular joint mechanism of the ankle E the rotator cuff. A sportsman complains of pain over A Musculotendinous junction tears of the the lateral side of his elbow. B Re-rupture of the Achilles tendon within A Common extensor origin 1 year of injury occurs in less than 10 per B Acromioclavicular joint cent of cases however they are treated.
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These two key results provide compelling evidence for a clustering hypothesis of large plaque formation and growth. Together, these data provide in vivo support for the clustering hypothesis by which clusters of small plaques merge together to form single plaques over time. This is a worrying prediction as the economic burden, in turns of palliative care and loss of economic contribution for so many patients and their carer-givers will be immense. Over the progression of the disease, memory, cognition and functional ability further diminish, frequently accompanied by symptoms of depression, anxiety and paranoia. Patients often die of concomitant secondary complications associated with inertia, such as pneumonia (Thies & Bleier, 2011). Neurofibrillary tangles are abnormal intracellular structures mainly consisting of hyperphosphorylated tau protein (Grundke-Iqbal et al. Tau is a microtubule stabilising protein that is normally bound to the microtubules and forms an integral part of the cell structure. What causes tau to be hyperphosphorylated and how amyloid (A) peptide might be involved is still a matter of debate. Amyloid plaques within the brain parenchyma are the main subject of this thesis and will be described in more detail in subsequent sections. As this cleavage occurs within the A amino acid stretch, A is not produced by this cleavage pathway (Figure 3b). A 42 has the greatest propensity to aggregate of all the A species as shown by in vitro experiments (Snyder et al. It is important to continue research into the physiological functions of these proteins as any therapy aimed at altering these cleavage pathways could have unintended side-effects. Mice are easy to breed and house and the technology to create transgenic mouse lines is well established. For example, neurofibrillary tangle pathology is only seen when an additional tau mutation (discovered in frontotemporal dementia patients) is expressed. Crossing the Tg2576 mouse with a mouse expressing Tau with the P301L mutation produced the same amount of plaques as Tg2576 mice, but the neurofibrillary tangle pathology was earlier and more extensive than the pure Tau P301L mouse (Lewis et al. Nevertheless, incomplete mouse models are very useful to investigate certain aspects of the disease, for example amyloid plaque development, in isolation without the added complication of comorbid pathology.
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Novel mecha nisms and approaches in the study of neurodegeneration and Ferruzzi, M. An in vivo model for the neurodegenerative ef fects of beta amyloid and protection by substance P. Neuroprotective effects of gallic acid against hy Memorias do Instituto Oswaldo Cruz, 103(5), 437-442. Gallic acid prevents memory defcits and oxidative stress induced by intracere Valizadeh, Z. Xi bao yu fen zi mian yi xue za zhi (Chinese Journal of Cellular and Molecular Immunology), 30(2), 155-159. Protective effects of gallic acid against Streptozotocin-induced oxidative damage Yang, H. Toona sinensis extracts induces ap optosis via reactive oxygen species in human premyelocytic Niho, N. Phytotherapy Research, 24(7), 1065 loid-induced cognitive dysfunction impairs glucose homeo 1070. Salidroside attenuates beta amyloid endophytes from Acer ginnala Maxim: isolation, identifca induced cognitive defcits via modulating oxidative stress tion and their yield of gallic acid. It also gives examples of setting up singing groups for people living in the community who are affected by dementia. Whilst hospitals may be used as a community singing venue, this guide is not concerned with specialised music therapy as a clinical intervention. Such work is undertaken by registered music therapists, often as part of a multi-disciplinary health team. Rather the guide draws upon the experience of groups of people who live with the effects of dementia and who sing in established, dementia-focused singing groups. It also draws on the experience of music practitioners and health researchers in the Sidney De Haan Research Centre for Arts and Health who have contributed significantly to the growing body of evidence and who have helped to establish singing groups for people with specific health conditions, including those affected by dementia. This guide is for anyone interested to setting up, running and evaluating community singing groups for the benefit of people living with dementia.
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