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Braak, Neuropathological stageing of Alzheimer-related changes, Acta 499 Neuropathol, 1991, 82, 239-259. Cidlowski, Uncoupling cell shrinkage from apoptosis reveals that Na+ 504 influx is required for volume loss during programmed cell death, J Biol Chem, 2003, 278, 505 39176-39184. Cidlowski, Plasma membrane depolarization 514 without repolarization is an early molecular event in anti-Fas-induced apoptosis, J Biol Chem, 515 2001, 276, 4304-4314. Choi, Mediation of neuronal apoptosis by enhancement of 522 outward potassium current, Science, 1997, 278, 114-117. Mennerick, Magnesium induces neuronal apoptosis by 524 suppressing excitability, Cell Death Dis, 2010, 1, e63. Goldberg, Ionized intracellular calcium 529 concentration predicts excitotoxic neuronal death: observations with low-affinity fluorescent 530 calcium indicators, J Neurosci, 1997, 17, 6669-6677. Schousboe, Excitatory amino acid-mediated cytotoxicity and calcium 534 homeostasis in cultured neurons, J Neurochem, 1993, 60, 1202-1211. Berry, Selenium and selenoproteins in the brain and brain diseases, J 536 Neurochem, 2003, 86, 1-12. Cooper, Selective divalent copper chelation for the treatment of diabetes mellitus, 557 Curr Med Chem, 2012, 19, 2828-2860. Shimizu, Brain-Specific Superoxide Dismutase 2 Deficiency Causes Perinatal Death 560 with Spongiform Encephalopathy in Mice, Oxid Med Cell Long, 2015, 2015, 238914. Mattson, Mitochondrial manganese 563 superoxide dismutase prevents neural apoptosis and reduces ischemic brain injury: 564 suppression of peroxynitrite production, lipid peroxidation, and mitochondrial dysfunction, the 565 J Neurosci, 1998, 18, 687-697. Hurley, Superoxide dismutase activity 567 and lipid peroxidation in the rat: developmental correlations affected by manganese deficiency, 568 J Nutr, 1983, 113, 2498-2504. Guilarte, Manganese neurotoxicity: lessons learned from longitudinal 570 studies in nonhuman primates, Environ Health Persp, 2009, 117, 325-332. Sunol, 572 Mechanisms of manganese-induced neurotoxicity in primary neuronal cultures: the role of 573 manganese speciation and cell type, Toxicological sciences: an official journal of the Society 574 of Toxicology, 2011, 124, 414-423. Miller, Zn2+ ions: modulators of excitatory and inhibitory 600 synaptic activity, Neuroscientist, 2004, 10, 432-442. Dringen, Metabolism and functions of copper in brain, Prog 608 Neurobiol, 2014, 116, 33-57. Kanaley, Increased regional brain 615 concentrations of ceruloplasmin in neurodegenerative disorders, Brain Res, 1996, 738, 265 616 274. Baker, Regeneration of the heart in diabetes by selective 635 copper chelation, Diabetes, 2004, 53, 2501-2508. Patronas, Neonatal diagnosis and treatment of Menkes disease, N Engl J Med, 645 2008, 358, 605-614. Llanos, Molecular and cellular aspects of copper transport in 647 developing mammals, J Nutr, 2003, 133, 1481S-1484S.
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Although there was a downtick in cost in 2012, not in the prescription ratio [186]. There is clear evidence that the early diagnosis of dementia benefts both health care professionals and carers [188]. Early diagnosis enables individuals to gain better access to information, resources, and support; demystify and destigmatize the condition; beneft from treatments; plan for the future; and prepare family, friends, and colleagues for the changes that can be expected. However, some doctors may not be confdent in their diagnosis in the early-onset period [189]. Normally it takes up to one year to diagnose and, therefore, it is a frustrating process for physicians, patients, and their relatives to diagnose and manage. Moreover, undetected dementia in younger people may be associated with work disability and an individual being (incorrectly) released from their job. Globally, dementia is a huge economic burden and is the fourth commonest cause of death among older people [194]. Moreover, dementia is still not widely recognized as a chronic condition that can impact every sector of society, and there is a commonly held belief that dementia is the disease of older people [195]. Nepal is a small, multi-ethnic, and multi-lingual (more than 68 languages) country. It is a low-income south Asian country with a population of about 30 million of which over 2. The demographic picture is one where the percentage of older people is growing faster than the total population. In terms of primary care, there are 268 physician-based primary health care clinics in the country. However, less than 1% of all health expenditure is directed towards mental health, and there is little investment in training for inpatient psychiatric unit staff (<2% for nurses and 2% doctors) and no community-based psychiatric inpatient facilities are available. All psychiatric issues, such as schizophrenia, neurotic disorders, anti-psychotic, antidepressant, mood stabilizer, anxiolytic, and antiepileptic cases, are diagnosed in the outpatient rooms in hospitals or in one of the central mental hospitals a total of 0. In Nepal, the total number of doctors was estimated to be 14,372 in 2013, as reported by the Nepal Medical Association. A majority of doctors are involved in the capital city of the country, and there are over 1000 specialized doctors in different sectors. In addition, there is a trend for Nepalese health care professionals to go aboard.
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In excitable cells such as neurons, outside the brain, consisting of afferent and sodium channels are responsible for the ris efferent nerves which gather information ing phase of action potentials as they change from and control the movements of the skele permeability and allow the exchange of tal muscles, as opposed to the brain and auto sodium and potassium ions, which causes nomic nervous system. The conversion of a psychological process to a bodily manifestation, as in the physio logical reactions and behaviors associated sodium pump (sodium-potassium pump) with anxiety up to development of ulcer and n. Within the neuron, an electrolytic balance heart disease as a result of chronic emotional must be attained for normal neural trans states. To obtain this electrical (ionic) and chemical balance, some sodium + somatization disorder ions (Na) must be relocated through the n. A family of psychological disturbances cell membrane from the intracellular por which produce bodily manifestations which tion of a neuron to the extracellular space by cause signifcant distress and interfere with an active mechanism, the sodium-potassium the daily functioning of the sufferer. The sodium-potas physical causes cannot be found and which sium pump exchanges intracellular sodium + are judged by the clinician to be due to psy ions for extracellular potassium ions (K), to chological factors; undifferentiated somato obtain the necessary cellular balance. A plant regarded as holy by sev without apparent physical cause and attrib eral ancient European societies. Of or relating to the body as distinct in which a person is incapacitated by multiple from the mind (psychic). Arising in the fears of having a disease based on misinterpre body or cells of the body, as in somatic tation of bodily symptoms that are not allayed mutations. It can be further divided into the cause signifcant distress and interfere with primary somatosensory area and somatosen the daily functioning of the sufferer. The primary somatosensory or other symptoms without obvious physical area receives information from the ventrolat cause and attributed by a clinician to psycho eral nucleus of the thalamus. Damage in this logical factors; conversion disorder, in which cortical area is associated with hypoesthesia, voluntary or sensory nerve functioning is two-point discrimination defects, and simi impaired (such as numbness or paralysis of a lar defcits in using somatosensory informa body part) without apparent physical cause tion. These cortical association areas further interferes with daily functioning and becomes process the somatosensory information. Any of the three basic body/mind types tation of bodily symptoms that are not allayed described by William Sheldon in his constitu by medical opinions to the contrary. Most of the postcentral gyrus in the front sound pressure level part of the parietal lobe, which is believed to n. Memory defect in which certain informa memory, spatial reasoning, navigation, spa tion can be recalled, but it is impossible to tial language, and mental rotation. Spatial recall the source or origin of that informa cognition is researched extensively in both tion.
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Atention in dichotic listening: Efective cues and tentional mechanisms in delay of gratifcation. Activit in right temporo-parietal junction is lateral prefontal cortex to the acquisition and extinction of not selective for theory-of-mind. The sovereignt of social cognition: Insights fom hemodynamic response in afective circuitry with aging: Novelt, neuroscience. Task-specifc signal transmission fom prefontal specifc efects of social cognition on the neural correlates of subse cortex in visual selective atention. A category-specifc response to animals medial prefontal contributions to judgments of similar and dissimi in the right human amygdala. Kinesthetic-visual matching and the self processing emotional facial expressions. Journal of Neurosci a high-level autistic subject with exceptional graphic abilities. The world around us: Neural command The neural system that mediates familiarit memory. Cost, beneft, tonic, phasic: What do response rates tell brain activit related to semantic and syntactic aspects of language. Schwartz, separation: A quantitative neuroimaging meta-analysis investigating & D. Neuronal activit in the younger and older adults: Exploring the characteristics of illusory primate premotor, supplementary, and precentral motor cortex dur memories. Decision making in and nonverbal social impairment in adolescent and adult males with patients with spinal cord damage: Aferent fedback and the autism. Channelrhodopsin-1: A light-gated resonance imaging of synesthesia: Activation of V4/V8 by spoken proton channel in gren algae. Forest before tres: The precedence of global features encoding and retrieval of events: What, where, and when. Access to information in working memory: cortex and the hippocampus to short-term memory: Evidence for a Exploring the focus of atention. How thinking controls feling: A social cognitive efects of auditory deprivation and acquisition of a visual language.